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    Elucidating protein function

    I ask that you refrains for pursuing questions about what I eat in the comment section. For many people it’s so overwhelming to contrast what they currently eat – probably a typical American diet of 500-600 daily grams of carbohydrates (200 grams of which are sugar) – with a diet of less than 50 daily grams of carbohydrate, which is what I consume.Remember, what I’m showing you here is what I have been eating for about the last 7 months.Alzheimer disease (AD) is a neurodegenerative disorder marked by cognitive and behavioral impairment that significantly interferes with social and occupational functioning.It is an incurable disease with a long preclinical period and progressive course.Note to readers: This post was written in December of 2011.PLEASE do not ask me why I eat ‘this’ or why I don’t eat ‘that’ — as what is shown here does not necessarily reflect what or how I eat today (or more importantly, how you should eat).When asking the question, “How much should I reduce my intake of carbohydrates? Do you have elevated blood glucose or triglycerides (these are determined from a standard blood test)? For the purpose of this question, even responding “yes” to one of these questions means you are predisposed to being insulin resistant. Consider this matrix, and let’s use me as an example.” it’s a good idea to start with two broader questions: There are technical ways to quantify the answer to the first question, which I will detail in future posts. As you can see, based on my poor genes and lofty goals, I find myself in the upper right square, which means I need to adopt the greatest amount of carbohydrate restriction.

    Human haploid DNA is 3-3.2x10^9 bp long, but compacted into 22 X and Y chromosomes.

    Any such long DNA free from any structural support gets broken during replication, recombination and transcription.

    That is the raison detre eukaryotic systems have designed to compact such long DNA in to compact threads called chromosomes.

    The following image depicts one of the cardinal neuroimaging findings in AD – hippocampal atrophy.

    A patient with preclinical AD may appear completely normal on physical examination and mental status testing.

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